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Supplier: TCI America
Description: CAS Number: 2050-60-4
MDL Number: MFCD00009442
Molecular Formula: C10H18O4
Molecular Weight: 202.25
Purity/Analysis Method: >99.0% (GC)
Form: Clear Liquid
Boiling point (°C): 246
Melting point (°C): -29
Flash Point (°C): 122
Specific Gravity (20/20): 0.99

SDS

Supplier: MilliporeSigma
Description: Cas Number 6153-56-6, Chemical Formula (Cooh)2 2 H2O, For Synthesis

SDS

Catalog Number: (76423-322)
Supplier: BIOASSAY SYSTEMS
Description: Direct determination of oxalate concentrations in biological samples.


Supplier: Thermo Scientific Chemicals
Description: MDL: MFCD00150039 Notes: - H2O at 200°C
Catalog Number: (75878-206)
Supplier: BIOASSAY SYSTEMS
Description: For quantitative determination of oxalate (oxalic acid) in urine and other samples.

Supplier: Ward's Science
Description: CAS Number: 62-76-0
Formula Weight: 134.00
Formula: (COONa)2
Hazard Info: Toxic
Density (g/mL): 2.34
Freezing Point (°C): 250-270
Synonyms: Ethanedioic Acid Disodium Salt
Shelf Life (months): 12
Storage: White

SDS

Catalog Number: (77439-320)
Supplier: Bioss
Description: Apical membrane anion-exchanger with wide epithelial distribution that plays a role as a component of the pH buffering system for maintaining acid-base homeostasis. Acts as a versatile DIDS-sensitive inorganic and organic anion transporter that mediates the uptake of monovalent anions like chloride, bicarbonate, formate and hydroxyl ion and divalent anions like sulfate and oxalate. Function in multiple exchange modes involving pairs of these anions, which include chloride-bicarbonate, chloride-oxalate, oxalate-formate, oxalate-sulfate and chloride-formate exchange. Apical membrane chloride-bicarbonate exchanger that mediates luminal chloride absorption and bicarbonate secretion by the small intestinal brush border membrane and contributes to intracellular pH regulation in the duodenal upper villous epithelium during proton-coupled peptide absorption, possibly by providing a bicarbonate import pathway. Mediates also intestinal chloride absorption and oxalate secretion, thereby preventing hyperoxaluria and calcium oxalate urolithiasis. Transepithelial oxalate secretion, chloride-formate, chloride-oxalate and chloride-bicarbonate transport activities in the duodenum are inhibited by PKC activation in a calcium-independent manner. The apical membrane chloride-bicarbonate exchanger provides also a major route for fluid and bicarbonate secretion into the proximal tubules of the kidney as well as into the proximal part of the interlobular pancreatic ductal tree, where it mediates electrogenic chloride-bicarbonate exchange with a chloride-bicarbonate stoichiometry of 1:2, and hence will dilute and alkalinize protein-rich acinar secretion. Mediates also the transcellular sulfate absorption and oxalate secretion across the apical membrane in the duodenum and the formate ion efflux at the apical brush border of cells in the proximal tubules of kidney.


Catalog Number: (76098-612)
Supplier: Bioss
Description: Apical membrane anion-exchanger with wide epithelial distribution that plays a role as a component of the pH buffering system for maintaining acid-base homeostasis. Acts as a versatile DIDS-sensitive inorganic and organic anion transporter that mediates the uptake of monovalent anions like chloride, bicarbonate, formate and hydroxyl ion and divalent anions like sulfate and oxalate. Function in multiple exchange modes involving pairs of these anions, which include chloride-bicarbonate, chloride-oxalate, oxalate-formate, oxalate-sulfate and chloride-formate exchange. Apical membrane chloride-bicarbonate exchanger that mediates luminal chloride absorption and bicarbonate secretion by the small intestinal brush border membrane and contributes to intracellular pH regulation in the duodenal upper villous epithelium during proton-coupled peptide absorption, possibly by providing a bicarbonate import pathway. Mediates also intestinal chloride absorption and oxalate secretion, thereby preventing hyperoxaluria and calcium oxalate urolithiasis. Transepithelial oxalate secretion, chloride-formate, chloride-oxalate and chloride-bicarbonate transport activities in the duodenum are inhibited by PKC activation in a calcium-independent manner. The apical membrane chloride-bicarbonate exchanger provides also a major route for fluid and bicarbonate secretion into the proximal tubules of the kidney as well as into the proximal part of the interlobular pancreatic ductal tree, where it mediates electrogenic chloride-bicarbonate exchange with a chloride-bicarbonate stoichiometry of 1:2, and hence will dilute and alkalinize protein-rich acinar secretion. Mediates also the transcellular sulfate absorption and oxalate secretion across the apical membrane in the duodenum and the formate ion efflux at the apical brush border of cells in the proximal tubules of kidney.


Supplier: Thermo Scientific Chemicals
Description: Applications: paint and varnish drier
Soluble in dilute acids. Slightly soluble in water
Catalog Number: (76709-588)
Supplier: AFG Bioscience
Description: Human Oxalate oxidase (OxO) ELISA Kit


Catalog Number: (76098-610)
Supplier: Bioss
Description: Apical membrane anion-exchanger with wide epithelial distribution that plays a role as a component of the pH buffering system for maintaining acid-base homeostasis. Acts as a versatile DIDS-sensitive inorganic and organic anion transporter that mediates the uptake of monovalent anions like chloride, bicarbonate, formate and hydroxyl ion and divalent anions like sulfate and oxalate. Function in multiple exchange modes involving pairs of these anions, which include chloride-bicarbonate, chloride-oxalate, oxalate-formate, oxalate-sulfate and chloride-formate exchange. Apical membrane chloride-bicarbonate exchanger that mediates luminal chloride absorption and bicarbonate secretion by the small intestinal brush border membrane and contributes to intracellular pH regulation in the duodenal upper villous epithelium during proton-coupled peptide absorption, possibly by providing a bicarbonate import pathway. Mediates also intestinal chloride absorption and oxalate secretion, thereby preventing hyperoxaluria and calcium oxalate urolithiasis. Transepithelial oxalate secretion, chloride-formate, chloride-oxalate and chloride-bicarbonate transport activities in the duodenum are inhibited by PKC activation in a calcium-independent manner. The apical membrane chloride-bicarbonate exchanger provides also a major route for fluid and bicarbonate secretion into the proximal tubules of the kidney as well as into the proximal part of the interlobular pancreatic ductal tree, where it mediates electrogenic chloride-bicarbonate exchange with a chloride-bicarbonate stoichiometry of 1:2, and hence will dilute and alkalinize protein-rich acinar secretion. Mediates also the transcellular sulfate absorption and oxalate secretion across the apical membrane in the duodenum and the formate ion efflux at the apical brush border of cells in the proximal tubules of kidney.


Catalog Number: (CAJTR769-1)
Supplier: AVANTOR PERFORMANCE MATERIAL LLC
Description: Formulation: 5.0 g/L New Methylene Blue N, 16.0 g/L potassium oxalate, monohydrate in water.

Catalog Number: (470301-549)
Supplier: Ward's Science
Description: CAS Number: 6153-56-6
Formula Weight: 126.07
Formula: HOOCCOOH·2H2O
Density (g/mL): 1.65
Boiling Point (°C): 149-160
Freezing Point (°C): 101.5
Solubility: Water and Organic Solvents
Synonyms: Ethanedioic Acid Dihydrate
Shelf Life (months): 36
Storage: White

SDS



Catalog Number: (10663-778)
Supplier: Bioss
Description: GOX is a 370 amino acid protein that is expressed in liver and pancreas. HAO1 is localized to peroxisomes and aids in organic acid metabolism via 2-hydroxyacid oxidase activity. 2-hydroxyacid oxidases, such as HAO1, are enzymes that require a flavin cofactor to oxidize 2-hydroxyacids to 2-ketoacids while reducing oxygen to hydrogen peroxide. HAO1 prefenentially oxidizes the substrate glycolate and also oxidizes other substrates, including 2-hydroxy fatty acids as well as L-?hydroxy acids of moderately short chain lengths. The oxidation of glycolate yields glyoxylate which is utilized for peroxisomal synthesis of glycine. HAO1 is also able to convert glyoxylate to oxalate. HAO1 is thought to play a role in the pathophysiology of hyperoxaluria type 1, which is caused by defects in AGXT, a peroxisomal enzyme, leading to accumulation of glyoxylate. Hyperoxaluria type 1 is characterized by an accumulation of oxalate that is thought to lead to precipitates of calcium oxalate in kidneys which can be fatal.


Supplier: Thermo Scientific Chemicals
Description: Applications: Metal polishing determination of pH
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