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Catalog Number: (CA10064-002)
Supplier: LONZA PHARMA - BIOSCIENCE CA
Description: Human Transferrin are iron-binding blood plasma glycoproteins that control the level of free iron in biological fluids. Human transferrin is encoded by the TF gene. Human Transferrin glycoproteins bind iron very tightly, but reversibly and are the most important iron pool, with the highest rate of turnover (25 mg/24 h).

Supplier: United Scientific Supplies
Description: Heavy cast-iron triangular bases.

Supplier: United Scientific Supplies
Description: Support rings for funnels of all sizes.

Catalog Number: (470355-408)
Supplier: American Scientific
Description: Prevent rust and eliminate the mess that comes with using iron filings.


Catalog Number: (10481-320)
Supplier: Bioss
Description: Serine protease which hydrolyzes a range of proteins including type I collagen, fibronectin and fibrinogen. Can also activate urokinase-type plasminogen activator with low efficiency. May play a specialized role in matrix remodeling processes in liver. Required to sense iron deficiency. Overexpression suppresses activation of the HAMP promoter.Involvement in disease:Defects in TMPRSS6 are the cause of iron-refractory iron deficiency anemia (IRIDA); also known as hypochromic microcytic anemia with defect in iron metabolism or hereditary iron-handling disorder or pseudo-iron-deficiency anemia. Key features include congenital hypochromic microcytic anemia, very low mean corpuscular erythrocyte volume, low transferrin saturation, abnormal iron absorption characterized by no hematologic improvement following treatment with oral iron, and abnormal iron utilization characterized by a sluggish, incomplete response to parenteral iron.


Supplier: Thermo Scientific Chemicals
Description: MDL: MFCD00078215 Notes: EDTA 70.2%
Supplier: Wards
Description: Heavy Retort Bases with tapped rod hole made of chemically-resistant coated cast iron.

Catalog Number: (10426-254)
Supplier: Bioss
Description: Stores iron in a soluble, non-toxic, readily available form. Important for iron homeostasis. Has ferroxidase activity. Iron is taken up in the ferrous form and deposited as ferric hydroxides after oxidation. Also plays a role in delivery of iron to cells. Mediates iron uptake in capsule cells of the developing kidney (By similarity).


Catalog Number: (10370-550)
Supplier: Bioss
Description: Important in metal transport, in particular iron. Can also transport manganese, cobalt, cadmium, nickel, vanadium and lead. Involved in apical iron uptake into duodenal enterocytes. Involved in iron transport from acidified endosomes into the cytoplasm of erythroid precursor cells. May play an important role in hepatic iron accumulation and tissue iron distribution. May serve to import iron into the mitochondria.


Supplier: Pace
Description: Size: 5.1x5.1mm. Shank Diameter: 0.48mm. For PS-90 Production Soldering Iron (30138-506). Can also be used with PACE* PS-80, SP-2A, and IR-70 irons.

Catalog Number: (75791-128)
Supplier: Prosci
Description: Ferritin heavy polypeptide 1(FTH1), is a ubiquitous intracellular protein which stores iron in a soluble, non-toxic, readily available form. FTH1 has ferroxidase activity and is important for iron homeostasis. Iron is taken up in the ferrous form and deposited as ferric hydroxides after oxidation. Ferritin is composed of 24 subunits of the light and heavy ferritin chains. It plays a role in delivery of iron to cells and mediates iron uptake in capsule cells of the developing kidney. Variation of ferritin subunit composition may affect iron absorption and release in different tissues. Deficiency of ferritin proteins may cause several neurodegenerative diseases. Almost all living organisms can produce this protein, including algae, bacteria, higher plants, and animals.


Supplier: Ward's Science
Description: CAS Number: 10028-22-5
Formula Weight: 562
Formula: Fe2(SO4)3·nH2O
Density (g/mL): 3.1
Solubility: Water
Synonyms: Ferric Sulfate, Monsel's Salt
Shelf Life (months): 12
Storage: Green

SDS

Catalog Number: (77578-508)
Supplier: Macherey-Nagel
Description: A pre-dosed NANOCOLOR® tube test for photometric determination of iron in water.

New Product


Supplier: Wards
Description: Individual cast iron slotted weights.

Catalog Number: (10481-336)
Supplier: Bioss
Description: Serine protease which hydrolyzes a range of proteins including type I collagen, fibronectin and fibrinogen. Can also activate urokinase-type plasminogen activator with low efficiency. May play a specialized role in matrix remodeling processes in liver. Required to sense iron deficiency. Overexpression suppresses activation of the HAMP promoter.Involvement in disease:Defects in TMPRSS6 are the cause of iron-refractory iron deficiency anemia (IRIDA); also known as hypochromic microcytic anemia with defect in iron metabolism or hereditary iron-handling disorder or pseudo-iron-deficiency anemia. Key features include congenital hypochromic microcytic anemia, very low mean corpuscular erythrocyte volume, low transferrin saturation, abnormal iron absorption characterized by no hematologic improvement following treatment with oral iron, and abnormal iron utilization characterized by a sluggish, incomplete response to parenteral iron.


Catalog Number: (10481-340)
Supplier: Bioss
Description: Serine protease which hydrolyzes a range of proteins including type I collagen, fibronectin and fibrinogen. Can also activate urokinase-type plasminogen activator with low efficiency. May play a specialized role in matrix remodeling processes in liver. Required to sense iron deficiency. Overexpression suppresses activation of the HAMP promoter.Involvement in disease:Defects in TMPRSS6 are the cause of iron-refractory iron deficiency anemia (IRIDA); also known as hypochromic microcytic anemia with defect in iron metabolism or hereditary iron-handling disorder or pseudo-iron-deficiency anemia. Key features include congenital hypochromic microcytic anemia, very low mean corpuscular erythrocyte volume, low transferrin saturation, abnormal iron absorption characterized by no hematologic improvement following treatment with oral iron, and abnormal iron utilization characterized by a sluggish, incomplete response to parenteral iron.


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