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Facial Tissues

Supplier: Dukal
Description: Facial tissues made of 100% virgin wood pulp.

New Product

Product Image-CA21903-003

KLEENEX® Facial Tissue, KIMBERLY-CLARK PROFESSIONAL®

Supplier: Kimberly-Clark
Description: When away from home, people naturally prefer the tissues they choose for themselves

Product Image-89140-690

About Face™ Cleansing Wipes, Micronova

Catalog Number: (89140-690)
Supplier: Micronova
Description: A pre-moistened cleansing wipe that is gentle enough for the face and that can be used to remove surface soil prior to gloving or sanitizing hands.

Small Business Enterprise Minority or Woman-Owned Business Enterprise


Product Image-10194-896

Schamberg Comedone Extractor, OR Grade, Sklar

Supplier: Sklar
Description: Sklar's Schamberg Comedone Extractor is a dermal instrument used to treat facial blemishes.

Product Image-76191-944

Comedone Extractor, OR-Grade, Sklar®

Catalog Number: (76191-944)
Supplier: Sklar
Description: This is a dermal instrument used to treat facial blemishes.
Product Image-10100-322

Anti-FUCA1 Rabbit Polyclonal Antibody

Catalog Number: (10100-322)
Supplier: Prosci
Description: Alpha-L-fucosidase (EC 3.2.1.51) is a lysosomal enzyme involved in the degradation of fucose-containing glycoproteins and glycolipids. At least 2 separate polymorphic alpha-L-fucosidases are recognized in man: that in tissues, FUCA1, which is deficient in fucosidosis, and that in plasma, FUCA2. Fucosidosis is an autosomal recessive lysosomal storage disease caused by defective alpha-L-fucosidase with accumulation of fucose in the tissues. Different phenotypes include clinical features such as neurologic deterioration, growth retardation, visceromegaly, and seizures in a severe early form; coarse facial features, angiokeratoma corporis diffusum, spasticity and delayed psychomotor development in a longer surviving form; and an unusual spondylometaphyseoepiphyseal dysplasia in yet another form.
Image Unavailable-10435-350

Anti-SLC2A10 Rabbit Polyclonal Antibody (Cy7®)

Catalog Number: (10435-350)
Supplier: Bioss
Description: Defects in SLC2A10 are the cause of arterial tortuosity syndrome (ATS) [MIM:208050]. ATS is an autosomal recessive disorder characterized by tortuosity and elongation of major arteries, often resulting in death at young age. Other typical features include aneurysms of large arteries and stenosis of the pulmonary artery, in association with facial features and several connective tissue manifestations such as soft skin and joint laxity. Histopathological findings include fragmentation of elastic fibers in the tunica media of large arteries.
Image Unavailable-10435-354

Anti-SLC2A10 Rabbit Polyclonal Antibody (HRP (Horseradish Peroxidase))

Catalog Number: (10435-354)
Supplier: Bioss
Description: Defects in SLC2A10 are the cause of arterial tortuosity syndrome (ATS) [MIM:208050]. ATS is an autosomal recessive disorder characterized by tortuosity and elongation of major arteries, often resulting in death at young age. Other typical features include aneurysms of large arteries and stenosis of the pulmonary artery, in association with facial features and several connective tissue manifestations such as soft skin and joint laxity. Histopathological findings include fragmentation of elastic fibers in the tunica media of large arteries.
Product Image-10435-332

Anti-SLC2A10 Rabbit Polyclonal Antibody

Catalog Number: (10435-332)
Supplier: Bioss
Description: Defects in SLC2A10 are the cause of arterial tortuosity syndrome (ATS) [MIM:208050]. ATS is an autosomal recessive disorder characterized by tortuosity and elongation of major arteries, often resulting in death at young age. Other typical features include aneurysms of large arteries and stenosis of the pulmonary artery, in association with facial features and several connective tissue manifestations such as soft skin and joint laxity. Histopathological findings include fragmentation of elastic fibers in the tunica media of large arteries.
Image Unavailable-10435-344

Anti-SLC2A10 Rabbit Polyclonal Antibody (Cy3®)

Catalog Number: (10435-344)
Supplier: Bioss
Description: Defects in SLC2A10 are the cause of arterial tortuosity syndrome (ATS) [MIM:208050]. ATS is an autosomal recessive disorder characterized by tortuosity and elongation of major arteries, often resulting in death at young age. Other typical features include aneurysms of large arteries and stenosis of the pulmonary artery, in association with facial features and several connective tissue manifestations such as soft skin and joint laxity. Histopathological findings include fragmentation of elastic fibers in the tunica media of large arteries.
Image Unavailable-10435-348

Anti-SLC2A10 Rabbit Polyclonal Antibody (Cy5.5®)

Catalog Number: (10435-348)
Supplier: Bioss
Description: Defects in SLC2A10 are the cause of arterial tortuosity syndrome (ATS) [MIM:208050]. ATS is an autosomal recessive disorder characterized by tortuosity and elongation of major arteries, often resulting in death at young age. Other typical features include aneurysms of large arteries and stenosis of the pulmonary artery, in association with facial features and several connective tissue manifestations such as soft skin and joint laxity. Histopathological findings include fragmentation of elastic fibers in the tunica media of large arteries.
Image Unavailable-10435-346

Anti-SLC2A10 Rabbit Polyclonal Antibody (Cy5®)

Catalog Number: (10435-346)
Supplier: Bioss
Description: Defects in SLC2A10 are the cause of arterial tortuosity syndrome (ATS) [MIM:208050]. ATS is an autosomal recessive disorder characterized by tortuosity and elongation of major arteries, often resulting in death at young age. Other typical features include aneurysms of large arteries and stenosis of the pulmonary artery, in association with facial features and several connective tissue manifestations such as soft skin and joint laxity. Histopathological findings include fragmentation of elastic fibers in the tunica media of large arteries.
Image Unavailable-10435-352

Anti-SLC2A10 Rabbit Polyclonal Antibody (FITC (Fluorescein Isothiocyanate))

Catalog Number: (10435-352)
Supplier: Bioss
Description: Defects in SLC2A10 are the cause of arterial tortuosity syndrome (ATS) [MIM:208050]. ATS is an autosomal recessive disorder characterized by tortuosity and elongation of major arteries, often resulting in death at young age. Other typical features include aneurysms of large arteries and stenosis of the pulmonary artery, in association with facial features and several connective tissue manifestations such as soft skin and joint laxity. Histopathological findings include fragmentation of elastic fibers in the tunica media of large arteries.
Image Unavailable-76118-104

Anti-GLUT10 Rabbit Polyclonal Antibody (Alexa Fluor® 680)

Catalog Number: (76118-104)
Supplier: Bioss
Description: Defects in SLC2A10 are the cause of arterial tortuosity syndrome (ATS) [MIM:208050]. ATS is an autosomal recessive disorder characterized by tortuosity and elongation of major arteries, often resulting in death at young age. Other typical features include aneurysms of large arteries and stenosis of the pulmonary artery, in association with facial features and several connective tissue manifestations such as soft skin and joint laxity. Histopathological findings include fragmentation of elastic fibers in the tunica media of large arteries.
Image Unavailable-76118-106

Anti-GLUT10 Rabbit Polyclonal Antibody (Alexa Fluor® 750)

Catalog Number: (76118-106)
Supplier: Bioss
Description: Defects in SLC2A10 are the cause of arterial tortuosity syndrome (ATS) [MIM:208050]. ATS is an autosomal recessive disorder characterized by tortuosity and elongation of major arteries, often resulting in death at young age. Other typical features include aneurysms of large arteries and stenosis of the pulmonary artery, in association with facial features and several connective tissue manifestations such as soft skin and joint laxity. Histopathological findings include fragmentation of elastic fibers in the tunica media of large arteries.
Image Unavailable-10474-936

Anti-CLPTM1 Rabbit Polyclonal Antibody

Catalog Number: (10474-936)
Supplier: Bioss
Description: Clefts of the oral-facial region usually occur in early fetal development and can affect the lip, the soft palate (the soft tissue in the back of the mouth) and the hard palate (the roof of the mouth). Cleft lip (with or without cleft palate) is a genetically complex birth defect that occurs in approximately one in every 750-1,000 live births. This is one of the most common birth defects and is multifactorial, with both genetic and environmental causes. Cleft lip- and palate-associated transmembrane protein 1 (CLPTM1) belongs to a family of cleft lip and palate transmembrane proteins. This family also contains cisplatin resistance-related protein (CRR9), which is involved in CDDP-induced apoptosis. The CLPTM1 protein shows strong homology to two Caenorhabditis elegans genes.
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Stock for this item is limited, but may be available in a warehouse close to you. Please make sure that you are logged in to the site so that available stock can be displayed. If the call is still displayed and you need assistance, please call us at 1-800-932-5000.
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